![]() ![]() Archers generally suck, so Khergits and Sarranids are not great at castle defense (mixed results against Vaegirs). Javelins are great but since their ammo count is pathetic javelineers are not the best ranged troops. The best archers are Rhodok Sharpshooters (in addition to high crossbow damage, they are actually good in melee and carry a huge shield also, since crossbows shoot slower they retain their ammunition longer in a battle rather than wasting it early).įor castle defense the Nords or Rhodoks are amazing (Rhodok Sharpshooters are hilariously-effective, so are Nord Huscarls) and in general, crossbowmen are lethal against all troops due to their high damage even at low levels and since walls help to cover their lack of armor. ![]() The best infantry is the Nord Huscarl (slowest-trained elite in the game, huge shield) followed by the Rhodok Sergeant (really good blunt weapon, heavy armor, gigantic shield). ![]() However the Khergit Lancer can be trained more quickly than either (fastest-trained elite unit in the game). Favorite cavalry are the Swadian Knight and Sarranid Mameluk. ![]()
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![]() EEG abnormalities are non-specific and indicate a genetic risk of epilepsy, but most patients do not have EEG slowing which is a correlate of encephalopathy. Seizure semiology is varied, with predominant myoclonic, astatic and absence seizure types with onset most commonly in infancy or early childhood. We highlight a significant risk of epilepsy in NGLY1 deficiency and detail the clinical and electrographic features identified in our international cohort. 7/17 subjects achieved complete seizure control on a variety of medications. Commonly used antiseizure medications were valproate, levetiracetam, lamotrigine, and clobazam. EEG background was otherwise normal without slowing in the majority of subjects. 10/14 (71%) subjects had generalized interictal epileptiform activity on EEG. Seizure types included myoclonic (8/17, 47%), astatic (7/17, 41%), and absence (6/17, 35%) focal with secondary generalization, tonic, generalized tonic clonic, and infantile spasms were also reported. 17/28 (60.7%) had a history of epilepsy, with mean seizure onset at 37 months (interquartile range 5–60 months, range 2 months to 19 years). Descriptive statistics are provided for the first year of an ongoing natural history study.Īll subjects had typical symptoms including: global developmental delay and/or intellectual disability, hypo- or alacrima, hyperkinetic movement disorder and transient elevation in transaminases. 14 subjects also underwent in-person evaluations including EEG, obtained via 20 lead standard array. Seizure and medication history was confirmed with prior records. ![]() We performed prospective phenotyping of 28 subjects with NGLY1 deficiency via standardized clinician interviews every 4 months of medical, developmental and seizure history. To refine the electroclinical phenotype of epilepsy in NGLY1 deficiency via prospective clinical and electroencephalogram (EEG) findings in an international cohort. ![]() Stanford University School of Medicine, Stanford, CA ![]() ![]() Full of destructions, violence, and a targeting system. ![]()
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